Developmental Exposure to Low Dose Xenoestrogens Alters Femur Length and Tensile Strength in Adult Mice.
All three of these compounds are present in common place sources like drinking water. Thus, it may be possible to alter adult bone length by manipulating the consumption of potential sources of these compounds. Estrogen operates best on height growth at an equilibrium quantity so we want to make sure that our estrogen is at just the right amount.
"Developmental DES exposure increased femur length 2.0% (t12 = -2.06, p = 0.031) in females and tended to increase femur length 1.3% (t9 = -1.03, p = 0.166) in males. Developmental BPA exposure, on the other hand, increased femur length 2.3% (t13 = -1.96, p = 0.036) in males and tended to increase femur length 1.0% in females (t14 = -0.84, p = 0.208) (Figure 2)."<-DES and BPA did not fail to increase femur length at the highest dose like EE2 so EE2 is the one you want to watch out for[EE2 is present in adult contraceptives]
However, "developmental exposure to 100 µg/kg DES, which is 1000 times greater than the dose used in the current study, resulted in shorter femurs that had increased total bone area and calcium content. These changes were the result of increased osteoblast activity as determined by an increased mineral apposition rate and decreased resorption rate due to a decrease in the number of osteoclast cells"<-Extremely high levels of DES does reduce height growth.
So what we can take from this is that when you're really young your levels of estrogen are too low to maximize height growth. Then when development occurs estrogen levels become too high thus you likely want to lower these levels.
So to maximize height growth:
Baby/Toddler->Raise Estrogen to reach Equilibrium.
After->Lower Estrogen Levels.
The problem is finding the right levels. Babies may not get the xenoestrogens they need to be tall from drinking water so other sources may be needed.
At some point, estrogen levels get too high for height growth so they have to be knocked down a little.